Flint Lab Publications

Selected Publications

Dehart CJ, Chahal JS, Flint SJ, Perlman DH.
Extensive post-translational modification of active and inactivated forms of endogenous p53.
Mol Cell Proteomics 2013 13: 1-17. | PubMed
Chahal JS, Flint SJ.
The p53 protein does not facilitate adenovirus type 5 replication in normal human cells.
J Virol. 2013 87: 6044-46. | PubMed
Chahal JS, Gallagher C, Dehart CJ, Flint SJ.
The repression domain of the E1B 55 kDa protein participates in countering interferon-induced inhibition of adenovirus replication.
J Virol. 2013 87: 4432-44. | PubMed
Kato SE, Chahal JS, Flint SJ.
Reduced infectivity of adenovirus type 5 particles and degradation of entering viral genomes associated with incomplete processing of the pre-terminal protein.
J Virol. 2012  86: 13554-65. | PubMed
Chahal JS, Qi J, Flint SJ.
The human adenovirus type 5 E1B 55 kDa protein obstructs inhibition of viral replication by type I interferon in normal human cells.
PLoS Pathog. 2012 8: e1002853. | PubMed
Kato SE, Huang W, Flint SJ.
Role of the RNA recognition motif of the E1B 55 kDa protein in the adenovirus type 5 infectious cycle.
Virology 2011 417(1):9-17. |  PubMed
Yatherajam G, Huang W, Flint SJ.
Export of adenoviral late mRNA from the nucleus requires the Nxf1/Tap export receptor.
J Virol. 2011 85: 1429-38.| PubMed
Miller DL, Rickards B, Mashiba M, Huang W, Flint SJ.
The adenoviral E1B 55-kilodalton protein controls expression of immune response genes but not p53-dependent transcription.
J Virol. 2009 83: 3591-603. | PubMed
Pérez-Berná AJ, Marabini R, Scheres SH, Menéndez-Conerjero R, Dmitriev IP, Curiel DT, Mangel WF, Flint SJ, San Martín C.
Structure and uncoating of immature adenovirus.
J. Mol. Biol. 2009 392(2):547-57. | PubMed
LeRoy G, Rickards B, Flint SJ.
The double bromodomain proteins Brd2 and Brd3 couple histone acetylation to transcription.
Mol Cell. 2008 30: 51-60. | PubMed
Miller DL, Myers CL, Rickards B, Coller HA, Flint SJ.
Adenovirus type 5 exerts genome-wide control over cellular programs governing proliferation, quiescence, and survival.
Genome Biol. 2007 8: R58. | PubMed